A potential role for nitric oxide pathway in tuberculous pleural effusion

ELGÜN ÜLKAR S., Kacmaz B., Durak I.

INTERNATIONAL JOURNAL OF TUBERCULOSIS AND LUNG DISEASE, cilt.9, sa.3, ss.339-343, 2005 (SCI-Expanded, Scopus) identifier identifier identifier

  • Yayın Türü: Makale / Tam Makale
  • Cilt numarası: 9 Sayı: 3
  • Basım Tarihi: 2005
  • Dergi Adı: INTERNATIONAL JOURNAL OF TUBERCULOSIS AND LUNG DISEASE
  • Derginin Tarandığı İndeksler: Science Citation Index Expanded (SCI-EXPANDED), Scopus
  • Sayfa Sayıları: ss.339-343
  • Anahtar Kelimeler: arginase, nitric oxide, tuberculosis, pleuritis, NECROSIS-FACTOR-ALPHA, PULMONARY TUBERCULOSIS, UP-REGULATION, SYNTHASE, EXPRESSION, NITROTYROSINE, METABOLISM, ARGINASE, ARGININE, MURINE
  • Ankara Üniversitesi Adresli: Evet

Özet

SETTING: Tuberculous pleural effusion leads to an immune response involving mainly immune and mesothelial cells. Nitric oxide (NO) produced by these cells may have antimycobacterial effects against Mycobacterium tuberculosis.