Apoptotic and necrotic death mechanisms are concomitantly activated in the same cell after cerebral ischemia

Creative Commons License

Unal-Cevik I., Kilinc M., Can A., Gursoy-Ozdemir Y., Dalkara T.

STROKE, cilt.35, sa.9, ss.2189-2194, 2004 (SCI-Expanded) identifier identifier identifier

  • Yayın Türü: Makale / Tam Makale
  • Cilt numarası: 35 Sayı: 9
  • Basım Tarihi: 2004
  • Doi Numarası: 10.1161/01.str.0000136149.81831.c5
  • Dergi Adı: STROKE
  • Derginin Tarandığı İndeksler: Science Citation Index Expanded (SCI-EXPANDED), Scopus
  • Sayfa Sayıları: ss.2189-2194
  • Anahtar Kelimeler: apoptosis, caspases, cathepsins, cerebral ischemia, focal, necrosis, SITU DNA FRAGMENTATION, NITRIC-OXIDE, NEURONAL APOPTOSIS, CASPASE ACTIVATION, ARTERY OCCLUSION, FOCAL ISCHEMIA, DISTINCT, BRAIN, DEPRIVATION, CATHEPSIN
  • Ankara Üniversitesi Adresli: Evet

Özet

Background and Purpose - Both necrotic and apoptotic cell death mechanisms are activated after cerebral ischemia. However, whether they are concomitantly active in the same cell or in discrete cell populations is not known.