WORLD NEUROSURGERY, cilt.97, ss.407-415, 2017 (SCI-Expanded)
BACKGROUND: The development of secondary brain injury via oxidative stress after traumatic brain injury (TBI) is well known. Decorin (DC) inactivates transforming growth factor beta 1, complement system, and tumor necrosis factor alpha, which are related to oxidative stress and apoptosis. Consequently, the aim of the present study was to evaluate the role of DC on TBI.