Hemidystonia secondary to cervical demyelinating lesions

Yucesan C., Tuncel D., AKBOSTANCI M. C., Yucemen N., Mutluer N.

EUROPEAN JOURNAL OF NEUROLOGY, cilt.7, sa.5, ss.563-566, 2000 (SCI-Expanded) identifier identifier identifier

  • Yayın Türü: Makale / Tam Makale
  • Cilt numarası: 7 Sayı: 5
  • Basım Tarihi: 2000
  • Doi Numarası: 10.1046/j.1468-1331.2000.t01-1-00120.x
  • Dergi Adı: EUROPEAN JOURNAL OF NEUROLOGY
  • Derginin Tarandığı İndeksler: Science Citation Index Expanded (SCI-EXPANDED), Scopus
  • Sayfa Sayıları: ss.563-566
  • Anahtar Kelimeler: hemidystonia, multiple sclerosis, spinal cord lesion, MULTIPLE-SCLEROSIS, PAROXYSMAL DYSTONIA, SPINAL-CORD, KINESIGENIC DYSTONIA, 1ST MANIFESTATION, INVOLVEMENT
  • Ankara Üniversitesi Adresli: Evet

Özet

Hemidystonia is usually associated with a structural lesion in the contralateral basal ganglia. We report a patient with definite multiple sclerosis, according to Poser's criteria, presenting with an acute-onset sustained left hemidystonia. Cranial T2-weighted magentic resonance imaging (MRI) showed several hyperintense lesions in the centri semiovali and in the periventricular area without basal ganglia involvement. Moreover cervical spinal cord T2-weighted MRI showed two hyperintense lesions in the left posterolateral spine at C2 and C3, and one lesion in the right posterolateral spine at C4 levels. The hemidystonia improved completely after daily treatment with 1000 mg of methylprednisolone, and cervical MRI was performed after the improvement which showed that the lesions had become smaller and less intense. Finally we consider that the hemidystonia may be caused by the cervical spinal cord lesions of multiple sclerosis.