Anatomical and pathological findings of renal aplasia and compensatory hypertrophy in a cat Bir kedide renal aplazi ve kompenzatorik hipertrofi olgusunda anatomik ve patolojik bulgular


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EKİM O., Bozkurt M. F., OTO Ç.

Ankara Universitesi Veteriner Fakultesi Dergisi, cilt.58, sa.4, ss.285-288, 2011 (SCI-Expanded) identifier

  • Yayın Türü: Makale / Tam Makale
  • Cilt numarası: 58 Sayı: 4
  • Basım Tarihi: 2011
  • Doi Numarası: 10.1501/vetfak_0000002489
  • Dergi Adı: Ankara Universitesi Veteriner Fakultesi Dergisi
  • Derginin Tarandığı İndeksler: Science Citation Index Expanded (SCI-EXPANDED), Scopus, TR DİZİN (ULAKBİM)
  • Sayfa Sayıları: ss.285-288
  • Anahtar Kelimeler: Agenesis, Aplasia, Cat, Hypertrophy, Kidney
  • Ankara Üniversitesi Adresli: Evet

Özet

In this study incidentally detected renal aplasia in right kidney and compensatory hypertrophy in left kidney have been defined with the anatomical and pathological findings in a 2 years old male crossbred cat which had suffered from infirmity, vomiting, anorexia and depression. In macroscopic examination, it was observed that the left kidney was located on the left side of the median line, at the ventral part of the transverse processes of the 1st - 4th lumbar vertebrae. The right kidney was at the right side of median line, ventral part of the transverse process of the 2nd lumbar vertebra. Left kidney had a yellow color and a firm consistency and also the cut sections were smooth. Besides, right kidney had a grayish-yellow color and the distinction between renal cortex and medulla in cut sections couldn't be distinguished. As of anatomical location and morphology, no abnormality was observed in the other organs of the cat. In right kidney, tubular structures were recognized histopathologically in between intense collagen tissue. Numerous of vascular formations were found near to the renal capsule. In hypertrophic kidney, in many of the tubules, degenerative alterations were observed significantly. Mcroscopically the most of the glomeruli were enlarged with mesangial cell hyperplasia and filled completely the Bowman's space. In the light of the detailed anatomical and pathological findings, the aim of this study was to make a contribution to such unilateral aplasia and compensatory hypertrophy cases that were rarely detected both in human and veterinary medicine.